Resistant bacterium are more widespread than previously thought, and may well be transmitting from person to person undetected. There are usually no symptoms at the time of initial infection. According to the US Centers for Disease Control and Prevention (CDC), the incidence of resistant bacterial infections are, indeed, on the rise.
With a marked decline in antibiotic effectiveness given new strains of resistant bacteria, the focus is now upstream on health optimization in order to maximize the body’s ability to recognize foreign invasion before the ‘tipping point’ of signs of infection. Attention to cellular health has never been more important considering current developments in infectious disease control.
Maintaining an effective immune response begins inside the cell. Mitochondrion, by definition, are the frontline cellular waste removal system. An infection of stealth bacteria can leave them weak, impotent and unable to perform the primary function of removing dangerous free radicals from the interior of the cell. A cascade of detrimental effects then follows, leaving the immune system, tissues and cells vulnerable to attacks by resistant, deadly bacteria.
The outer membrane of the mitochondrion have channels or narrow holes. Protein is too large to cross through the hole. But when such protein is destroyed, for example unfolds, it can easily fall through the hole or “trash chute”. The channels, through which proteins are removed, are also used to transport protein into the mitochondria. Blockage or infection of these channels results in toxic buildup and disease.
Cell injury and death happen through depolarization of the mitochondrial inner membrane – through some sort of stress, either external or internal – causing free radicals to be generated. These free radicals release cytochrome C into the cellular fluid, which sets into motion a cascade of enzymes that slice up and dispose of the cell.
Since mitochondria have their own genes, they are subject to mutations from toxins and stealth bacteria that affect their health and function. These mutations affect the way the cell functions. Mutations and toxic overload is the beginning of cellular and immune dysfunction leading to systemic infections and disease.
Some of these mitochondrial diseases might not become apparent until the person with infected mitochondrion reaches a certain age. A young muscle cell can have 85% of its mitochondria mutation-free, meaning it can handle all of the energy demands placed on it. Keep in mind, as the number of mitochondria decline with age, the energy demands placed on the remaining mitochondria rise resulting in the cascade of a disease process. Faster aging, decreased energy and impaired immune function are the result.
There comes a point where the mitochondrion can’t produce enough energy and the affected organ or organs start to display diminished capacity. Maintaining a high degree of normal, healthy mitochondria could well eliminate many of the diseases related to aging.
By upregulating mitochondria through BXprotocol, the cell is, in effect, reversing damage that otherwise leads to cell injury and death as described above. Keeping the mitochondrion “clean” is a similar action to changing the air filter in your home or car, preventing the leakage of toxins into the surrounding cellular environment.
By restoring optimal mitochondrial function, the immune and cellular toxin removal systems are better prepared to perceive and eliminate threats from deadly bacteria transmitted in everyday life and person-to- person interaction. There is no better “health insurance” than optimizing and upregulating mitochondrial function.