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Mitochondrial dysfunction and glucose intolerance was first described over 40 years ago. Many research studies now describe how mitochondrial dysfunction exhibits lower oxidative enzyme activities and decreased fat metabolism in muscle.

In simple terms, the ability to use fat and lose fat becomes impaired

A metabolically healthy environment is one that enables the body to burn fat as fuel and release fat from between muscle fibers when exercising. If your client is not seeing results from exercise and healthy eating, mitochondria may be to blame.

In healthy adults, exercise and calorie restriction are usually effective at improving overweight and obesity. You may want to look at mitochondrial efficiency if losing weight is difficult or impossible for your client.

Over the last 20 years, researchers have begun to identify changes in mitochondrial function when it comes to overweight and obesity.

Mitochondria control ATP production, energy expenditure, and disposal of ROS (reactive oxygen species). When mitochondria have an excessive work load, this leads to impaired fat and glucose metabolism.

Addressing the underlying “excessive work load” of mitochondria becomes of primary importance when looking at long term solutions to overweight and obesity.

Consider BX Protocol™ a first line solution for clients who aren’t getting results from exercise and healthy eating alone.[/fusion_text][fusion_text]Contrary to popular belief, Lorem Ipsum is not simply random text. It has roots in a piece of classical Latin literature from 45 BC, making it over 2000 years old. Richard McClintock, a Latin professor at Hampden-Sydney College in Virginia, looked up one of the more obscure Latin words

Curr Opin Endocrinol Diabetes Obes. 2010 Oct; 17(5): 446–452. doi:  10.1097/MED.0b013e32833c3026
PMCID: PMC5001554
Mitochondrial Dysfunction in Obesity
Juan C. Bournat1 and Chester W. Brown1,2,3

Endocr Connect. 2015 Mar 1; 4(1): R1–R15.
Published online 2014 Dec 9. doi:  10.1530/EC-14- 0092
PMCID: PMC4261703
Mitochondrial dysfunction and insulin resistance: an update
Magdalene K Montgomery and Nigel Turner